In a farm of geese with an approximate census of 1500 animals in the locality of Almonte, an abnormal increase in mortality was detected in December (>10% in 24 hours). Some birds showed lethargy and anorexia, respiratory difficulty, cough, and nasal discharge, and swollen sinuses while others showed diarrhea. Within a few days, a very high increase in the number of affected birds was observed, with several of them also showing neurological signs (tremors, convulsions, paralysis) y. These clinical signs were observed in animals of all ages.
In the post-mortem examination, the following signs and lesions were observed: congestion and edema of the airways, pancreatitis, necrotic foci in the spleen, liver, and kidneys, enteritis, congestive internal organs, edema in the head and neck, cerebral hemorrhage. Histologically, edema and perivascular cuffs were observed in the myocardium, spleen, and lungs, and gliosis, vascular proliferation, and neuronal degeneration in the brain.
Samples of blood, tracheal swab, spleen, lungs, and feces were taken and sent to the laboratory for analysis.
A cell culture and a rapid hemagglutination test on a slide were performed, revealing that it is a hemagglutinating pathogen.
In the laboratory, PCR and identification sequencing are carried out on different samples. An enveloped RNA virus that codes for a hemagglutinin and a neuraminidase is detected. In addition, the intravenous pathogenicity index is evaluated in 6 week-old chicks. IVPI=2